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【下載完整報告】AI熱潮從學術研究也能看出端倪?哪些議題是2023熱搜議題?
期刊
Rosiglitazone Restores Endothelial Dysfunction in a Rat Model of Metabolic Syndrome through PPARγ- and PPARδ-Dependent Phosphorylation of Akt and eNOS
Zhigang Zhao
;
Zhidan Luo
;
Peijian Wang
;
Jing Sun
;
Hao Yu
;
Tingbing Cao
;
Yinxing Ni
;
Jing Chen
;
Zhencheng Yan
;
Daoyan Liu
;
Zhiming Zhu
《PPAR Research》
2011卷
(2011/12)
Pp. 62-70
https://doi.org/10.1155/2011/291656
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Koch, E., Pircher, J., Czermak, T., Gaitzsch, E., Alig, S., Mannell, H., Niemeyer, M., Krötz, F., & Wörnle, M. (2013).
The Endothelial Tyrosine Phosphatase SHP-1 Plays an Important Role for Vascular Haemostasis in TNFα-Induced Inflammation In Vivo
.
Mediators of Inflammation
,
2013
(), 842-852-074. https://doi.org/10.1155/2013/279781
Chiu, H. M., Lin, H. H., & Tang, M. J. (1998).
Ethyl isopropylamiloride downregulates na,K-ATPase Gene expression which confers cytotoxicity in primary proximal tubule cell cultures
.
The Chinese Journal of Physiology
,
41
(4), 195-202. https://www.airitilibrary.com/Article/Detail?DocID=03044920-199812-41-4-195-202-a
Yang, C. (2010).
Roles of prostaglandin E2 receptors and chloride channels in epoxyeicosatrienoic acids-induced relaxation in rat mesentericarteries
[doctoral dissertation, The University of Hong Kong]. Airiti Library. https://www.airitilibrary.com/Article/Detail?DocID=U0029-1812201200018255
Souza, M. C., Paixão, F. H. M., Ferraris, F. K., Ribeiro, I., & Henriques, M. D. G. M. O. (2012).
Artesunate Exerts a Direct Effect on Endothelial Cell Activation and NF-κB Translocation in a Mechanism Independent of Plasmodium Killing
.
Malaria Research and Treatment
,
2012
(), 48-59. https://doi.org/10.1155/2012/679090
Liu, Y., Tian, X. Y., Huang, Y., & Wang, N. (2014).
Rosiglitazone Attenuated Endothelin-1-Induced Vasoconstriction of Pulmonary Arteries in the Rat Model of Pulmonary Arterial Hypertension via Differential Regulation of ET-1 Receptors
.
PPAR Research
,
2014
(), 67-72. https://doi.org/10.1155/2014/374075
國際替代計量
Rosiglitazone Restores Endothelial Dysfunction in a Rat Model of Metabolic Syndrome through PPARγ- and PPARδ-Dependent Phosphorylation of Akt and eNOS
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