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【下載完整報告】AI熱潮從學術研究也能看出端倪?哪些議題是2023熱搜議題?
期刊
AICAR Protects against High Palmitate/High Insulin-Induced Intramyocellular Lipid Accumulation and Insulin Resistance in HL-1 Cardiac Cells by Inducing PPAR-Target Gene Expression
Ricardo Rodríguez-Calvo
;
Manuel Vázquez-Carrera
;
Luis Masana
;
Dietbert Neumann
《PPAR Research》
2015卷
(2015/12)
Pp. 188-199
https://doi.org/10.1155/2015/785783
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延伸閱讀
Zhang, X., Zhu, P., Zhang, X., Ma, Y., Li, W., Chen, J. M., Guo, H. M., Bucala, R., Zhuang, J., & Li, J. (2013).
Natural Antioxidant-Isoliquiritigenin Ameliorates Contractile Dysfunction of Hypoxic Cardiomyocytes via AMPK Signaling Pathway
.
Mediators of Inflammation
,
2013
(), 198-207-118. https://doi.org/10.1155/2013/390890
胡家齊(2017)。
Indoleamine 2,3-Dioxygenase Promotes HCC Cell Proliferation by Activating β-catenin Signaling Through Aryl Hydrocarbon Receptor Pathway
〔碩士論文,國立臺灣大學〕。華藝線上圖書館。https://doi.org/10.6342/NTU201702296
Chang, C. C. (2013).
Molecular Mechanisms underlying Aryl-Hydrocarbon Receptor-Mediated Alterations in Angiogenesis and Vascular Integrity via RhoA Activation in Vascular Endothelial Cells
[doctoral dissertation, Taipei Medical University]. Airiti Library. https://doi.org/10.6831/TMU.2013.00002
Colon, E. G., Sampson, R. D., Sattler, S., Schneider, M. D., Rosenthal, N., & Tonkin, J. (2015).
Cardiac-Restricted IGF-1Ea Overexpression Reduces the Early Accumulation of Inflammatory Myeloid Cells and Mediates Expression of Extracellular Matrix Remodelling Genes after Myocardial Infarction
.
Mediators of Inflammation
,
2015
(), 384-393-235. https://doi.org/10.1155/2015/484357
Beyrich, C., Löffler, J., Kobsar, A., Speer, C. P., Kneitz, S., & Eigenthaler, M. (2011).
Infection of Human Coronary Artery Endothelial Cells by Group B Streptococcus Contributes to Dysregulation of Apoptosis, Hemostasis, and Innate Immune Responses
.
Mediators of Inflammation
,
2011
(), 491-498. https://doi.org/10.1155/2011/971502
國際替代計量
AICAR Protects against High Palmitate/High Insulin-Induced Intramyocellular Lipid Accumulation and Insulin Resistance in HL-1 Cardiac Cells by Inducing PPAR-Target Gene Expression
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延伸閱讀
國際替代計量
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