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【下載完整報告】AI熱潮從學術研究也能看出端倪?哪些議題是2023熱搜議題?
期刊
Mycophenolic Acid Overcomes Imatinib and Nilotinib Resistance of Chronic Myeloid Leukemia Cells by Apoptosis or a Senescent-Like Cell Cycle Arrest
Claire Drullion
;
Valérie Lagarde
;
Romain Gioia
;
Patrick Legembre
;
Muriel Priault
;
Bruno Cardinaud
;
Eric Lippert
;
François-Xavier Mahon
;
Jean-Max Pasquet
《Leukemia Research and Treatment》
2012卷
(2012/12)
Pp. 190-198
https://doi.org/10.1155/2012/861301
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Moraes, G. N. D., Souza, P. S., Costas, F. C. D. F., Vasconcelos, F. C., Reis, F. R. S., & Maia, R. C. (2012).
The Interface between BCR-ABL-Dependent and -Independent Resistance Signaling Pathways in Chronic Myeloid Leukemia
.
Leukemia Research and Treatment
,
2012
(), 151-169. https://doi.org/10.1155/2012/671702
Ryningen, A., Reikvam, H., Nepstad, I., Rye, K. P., & Bruserud, Ø. (2012).
Inhibition of Mammalian Target of Rapamycin in Human Acute Myeloid Leukemia Cells Has Diverse Effects That Depend on the Environmental In Vitro Stress
.
Bone Marrow Research
,
2012
(), 106-115. https://doi.org/10.1155/2012/329061
Huang, T. C., Hung, H. M., Chang, P. Y., Dai, M. S., Ho, C. L., Chen, Y. C., Chao, T. Y., & Kao, W. Y. (2012).
Long-term Follow-up Results of Chronic Myeloid Leukemia by RQ-PCR Monitoring of BCR-ABL Transcripts in Imatinib Era-A Single Institutional Experience
.
Journal of Medical Sciences
,
32
(6), 271-278. https://doi.org/10.6136/JMS.2012.32(6).271
Wu, Y., Qiu, H., Zeng, Y., You, X., Deng, Z., Yu, M., & Zhu, C. (2008).
Mycoplasma genitalium Lipoproteins Induce Human Monocytic Cell Expression of Proinflammatory Cytokines and Apoptosis by Activating Nuclear Factor κB
.
Mediators of Inflammation
,
2008
(), 71-78. https://doi.org/10.1155/2008/195427
黃郁雯(2011)。
Differentiation-inducing Agents Sensitize Human Chronic Myeloid Leukemia Cell Line K562 to STI571-Induced Apoptosis and Their Molecular Mechanisms
〔碩士論文,臺北醫學大學〕。華藝線上圖書館。https://www.airitilibrary.com/Article/Detail?DocID=U0007-2907201100141400
國際替代計量
Mycophenolic Acid Overcomes Imatinib and Nilotinib Resistance of Chronic Myeloid Leukemia Cells by Apoptosis or a Senescent-Like Cell Cycle Arrest
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