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【下載完整報告】AI熱潮從學術研究也能看出端倪?哪些議題是2023熱搜議題?
期刊
Paraoxonase-1 Inhibits Oxidized Low-Density Lipoprotein-Induced Metabolic Alterations and Apoptosis in Endothelial Cells: A Nondirected Metabolomic Study
Anabel García-Heredia
;
Judit Marsillach
;
Anna Rull
;
Iris Triguero
;
Isabel Fort
;
Bharti Mackness
;
Michael Mackness
;
Diana M. Shih
;
Jorge Joven
;
Jordi Camps
《Mediators of Inflammation》
2013卷
(2013/12)
Pp. 226-234-021
https://doi.org/10.1155/2013/156053
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Germain, N., Corbel, M., Belleguic, C., Boichot, E., & Lagente, V. (2001).
Effects of PDE4 inhibitors on lipopolysaccharide-induced priming of superoxide anion production from human mononuclear cells
.
Mediators of Inflammation
,
2001
(), 117-123. https://doi.org/10.1080/09629350120072680
Shirato, K., & Imaizumi, K. (2014).
Posttranscriptional Suppression of Lipopolysaccharide-Stimulated Inflammatory Responses by Macrophages in Middle-Aged Mice: A Possible Role for Eukaryotic Initiation Factor 2����
.
International Journal of Inflammation
,
(2014), 55-66. https://doi.org/10.1155/2014/292986
Daher, J., Martin, M., Rousseau, A., Nuyens, V., Kazan, H. F., Antwerpen, P. V., Courbebaisse, G., Martiat, P., Badran, B., Dequiedt, F., Boudjeltia, K. Z., & Vanhamme, L. (2014).
Myeloperoxidase Oxidized LDL Interferes with Endothelial Cell Motility through miR-22 and Heme Oxygenase 1 Induction: Possible Involvement in Reendothelialization of Vascular Injuries
.
Mediators of Inflammation
,
(2014-1), 138-151. https://doi.org/10.1155/2014/134635
詹景勛(2012)。
The Inhibitory Mechanisms of Radicicol on Lipopolysaccharide-induced Inflammatory Cytokine and Matrix Metalloproteinase-9 Expression in Human Monocytic Cells
〔碩士論文,臺北醫學大學〕。華藝線上圖書館。https://doi.org/10.6831/TMU.2012.00166
Eren, E., Yilmaz, N., & Aydin, O. (2013).
Functionally Defective High-Density Lipoprotein and Paraoxonase: A Couple for Endothelial Dysfunction in Atherosclerosis
.
Cholesterol
,
2013
(), 89-98. https://doi.org/10.1155/2013/792090
國際替代計量
Paraoxonase-1 Inhibits Oxidized Low-Density Lipoprotein-Induced Metabolic Alterations and Apoptosis in Endothelial Cells: A Nondirected Metabolomic Study
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