並列摘要
The passage of meconium in utero is either a normal event or more commonly associated with fetal asphyxia and decreased umbilical venous PO2. Fetal intestinal ischemia is augmented by the diving reflex, which shunts blood preferentially to the brain and away from the visceral organs leading to the passage of meconium. During fetal distress, the gasping efforts of the fetus are contribute to the entery of meconium into the respiratory tract. Clinical symptoms of meconium aspiration may be very mild, but typically the infant presents with tachypnea, prolonged expiratory phase and hypoxia shortly after birth. Air leak and pulmonary hypertension are often occurred in severe cases. Postnatal therapy of meconium aspiration starts with monitoring infants at risk. Chest physiotherapy and suctioning may be valuable and correction of hypoxemia and acidosis for pulmonary vasoconstrition is critical.
並列關鍵字
無資料延伸閱讀
- 盧建甫、黃富源（1980）。新生兒胎便吸入症候群。當代醫學，(83)，820-823。https://doi.org/10.29941/MT.198009.0010
- 滕儒錚、呂立、吳宗瑾、江伯倫（1996）。胎便吸入症候群。當代醫學，(275)，707-711。https://doi.org/10.29941/MT.199609.0006
- Lin, H. C., Su, B. H., Lin, T. W., Peng, C. T., & Tsai, C. H. (2004). 胎便吸入症候群的死亡危險因子：314病例分析. Acta Paediatrica Taiwanica, 45(1), 30-34. https://doi.org/10.7097/APT.200402.0030
- 楊俊榮、楊寶麟（1991）。胎便性腹膜炎。當代醫學，(213)，556-559。https://doi.org/10.29941/MT.199107.0005
- 許永祥、郭明勳、蘇益仁、候書文（1991）。疑似胎便腹膜炎。慈濟醫學雜誌，3(4)，327-330。https://doi.org/10.6440/TZUCMJ.199112.0327