Movement disorder is often caused by lesion in the contralateral basal ganglia. This paper reports an old woman with hemiballism-hemichorea induced by non-ketotic hyperglycemia and her neuroimaging studies presenting with an ipsilateral lesion. A brain computed tomography revealed a hyperdense lesion in the ipsilateral putamen, while magnetic resonance image study revealed an abnormal signal change in the ispilateral putamen; hyperintensity on Ti-weighted and hypointensity on T2-weighted images. She was treated with insulin to control her hyperglycemia, and fellow-up brain CT 4 months later showed complete resoluation of the abnormal attenuation, and no recurrent episodes of hyperkinesia was noted.
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