- 期刊
The Role of Heat Shock Protein 27/60 in Periodontitis and Cardiovascular Disease
並列摘要
When subjected to physiological stress such as heat stress or chemical stimulation, cells produce heat shock proteins (HSPs), which play an important role in coping with the stress and protecting cells from its harmful effects. HSPs act as regulators of apoptosis, either in the FasL or the tumor necrosis factor (TNF)-α. pathway. Apoptosis regulated by HSPs also has an effect on inflammation of periodontal tissues, and many studies have proven that HSPs are closely associated with cardiovascular disease. In recent years, the link between periodontal disease and cardiovascular disease has gradually been established. This article reviews the roles of HSPs in apoptosis, periodontitis, and cardiovascular disease.
延伸閱讀
- 陳惠華、曾春典、袁國、曾春褀（2005）。Current Status on the Relationship between Periodontal Disease and Cardiovascular Disease。中華民國牙周病醫學會雜誌，10(1)，77-89。https://www.airitilibrary.com/Article/Detail?DocID=10279962-200503-10-1-77-89-a
- Dillmann, W. (1999). Heat Shock Proteins and Protection Against Ischemic Injury. Infectious Diseases in Obstetrics and Gynecology, 1999(), 55-57. https://doi.org/10.1155/S1064744999000113
- Lee, H. J., Ock, C. Y., Kim, S. J., & Hahm, K. B. (2010). Heat Shock Protein: Hard Worker or Bad Offender for Gastric Diseases. International Journal of Proteomics, 2010(), 2-12. https://doi.org/10.1155/2010/259163
- 張采宇（2009）。The study of heat shock protein 47 in cyclosporin A induced gingival overgrowth〔碩士論文，中山醫學大學〕。華藝線上圖書館。https://doi.org/10.6834/CSMU.2009.00146
- 董醒任（1998）。Periodontal Disease and Cardiovascular Disease。中華民國牙周病醫學會雜誌，3(1)，50-50。https://www.airitilibrary.com/Article/Detail?DocID=a0000071-199803-201212270006-201212270006-50