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運動對腹部脂肪質量及胰島素敏感活性的影響

The Effects of Exercise on Visceral Fat and Insulin Sensitivity

摘要


II 型糖尿病的發生,主要是因為胰島素阻抗所造成的。至於造成胰島素阻抗的真正原因,似乎還不是很明確。最近研究發現脂締素與脂肪代謝及全身性胰島素敏感活性成正相關。脂締素主要從脂肪細胞產生,脂肪細胞的健康影響了脂締素的分泌。當脂肪組織質量越大或脂肪細胞體積越大時,脂締素的分泌反而越少。因為腹部脂肪質量更能有效預估血液脂締素的濃度,減少腹部脂肪的質量,能增加血漿脂締素濃度,減少胰島素阻抗性,改善II型糖尿病患高血糖及高血脂的問題。本研究的目的是透過文獻探討的方式,更深入了解,為何腹部肥胖與血漿脂締素的關係較為密切;另外不同運動型式對減少腹部肥胖的影響也有所討論。研究發現高強度運動,或結合阻力訓練的有氧訓練,似乎更能有效減少腹部脂肪質量,增加全身胰島素敏感活性。

並列摘要


Type II diabetes is mainly caused by insulin resistance, but the exact cause of insulin resistance is still not fully understood. Recent research found that there was a positive correlation between adiponectin and insulin sensitivity. Adiponectin mainly produced by adipose tissue, and the health status of adipose tissue have a significant influence on adiponectin production. When the mass of adipose tissue or the size of fat cell increased, the production of adiponectin was decreased. Since visceral fat deposits is more reliable in predicting blood adiponectin concentration, a decrease in visceral fat mass can increase plasma adiponectin concentration and increase insulin sensitivity and decrease blood sugar and lipid concentration on type II diabetes patients. By reviewing of literatures, the purpose of this research was to have a more thorough understanding on why there was a close relationship between visceral fat mass and plasma adiponectin concentration; also, the effects of different type of exercise training on visceral fat mass reduction will be discussed. Research found that high intensity exercise or a combining of resistance training and aerobic training is more effectively in reducing visceral fat mass and increasing insulin sensitivity.

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