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【下載完整報告】AI熱潮從學術研究也能看出端倪?哪些議題是2023熱搜議題?
期刊
Alpha 1-Antichymotrypsin, an Inflammatory Protein Overexpressed in the Brains of Patients with Alzheimer's Disease, Induces Tau Hyperphosphorylation through c-Jun N-Terminal Kinase Activation
Ethika Tyagi
;
Tina Fiorelli
;
Michelle Norden
;
Jaya Padmanabhan
《International Journal of Alzheimer's Disease》
2013卷
(2013/12)
Pp. 119-129
https://doi.org/10.1155/2013/606083
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Bórquez, D. A., & Billault, C. G. (2012).
The Amyloid Precursor Protein Intracellular Domain-Fe65 Multiprotein Complexes: A Challenge to the Amyloid Hypothesis for Alzheimer's Disease?
.
International Journal of Alzheimer's Disease
,
2012
(), 259-268. https://doi.org/10.1155/2012/353145
Bie, J. D., Lim, C. K., & Guillemin, G. J. (2016).
Progesterone Alters Kynurenine Pathway Activation in IFN-γ-Activated Macrophages - Relevance for Neuroinflammatory Diseases
.
International Journal of Tryptophan Research
,
(2016), 89-93. https://doi.org/10.4137/IJTR.S40332
Weng, P. H. (2011).
The Association between CHRNA7 Polymorphisms and Cognitive Response to Acetylcholinesterase Inhibitors in Alzheimer’s Disease
[master's thesis, National Taiwan University]. Airiti Library. https://doi.org/10.6342/NTU.2011.01848
Ni, Z., & Dong, H. (2021).
Inhibition of Amyloid-β Induced Signaling as an Approach to Prevent Neurofibrillary Tangle Formation and Cure Alzheimer's Disease
.
International Core Journal of Engineering
,
7
(1), 195-200. https://doi.org/10.6919/ICJE.202101_7(1).0026
Na, H. K., & Surh, Y. J. (2008).
The Antitumor Ether Lipid Edelfosine (ET-18-O-CH3) Induces Apoptosis in H-ras Transformed Human Breast Epithelial Cells: By Blocking ERK1/2 and P38 Mitogenactivated Protein Kinases as Potential Targets
.
Asia Pacific Journal of Clinical Nutrition
,
17
(S1), 204-207. https://doi.org/10.6133/apjcn.2008.17.s1.49
國際替代計量
Alpha 1-Antichymotrypsin, an Inflammatory Protein Overexpressed in the Brains of Patients with Alzheimer's Disease, Induces Tau Hyperphosphorylation through c-Jun N-Terminal Kinase Activation
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