To elucidate the refractive status of strabismic patients, we analyze the cycloplegic refractive errors of 521 divergent strabismics, 535 convergent strabismics, 32 congenital esotropes and 68 verticals. The distribution of refractive status of exotropes and vertical strabismics is analogous to that of normal population with comparable ages. Convergent strabismics, however, have relatively less myopic refractive errors. In horizontal heterotropia, there is a less ametropia in the fixating eyes (p<0.05), i. e. less myopic in exotropes and less hyperopic in esotropes. The prevalence of high anisometropia (difference in spherical equivalence between the two eyes of more than 2D) and astigmatism (astigmatism of more than 2D) increases as the eyes become more ametropic, especially prominent in exotropes. The presence of anisometropia and astigmatism may play a role in the pathogenesis of heterotropia.
To elucidate the refractive status of strabismic patients, we analyze the cycloplegic refractive errors of 521 divergent strabismics, 535 convergent strabismics, 32 congenital esotropes and 68 verticals. The distribution of refractive status of exotropes and vertical strabismics is analogous to that of normal population with comparable ages. Convergent strabismics, however, have relatively less myopic refractive errors. In horizontal heterotropia, there is a less ametropia in the fixating eyes (p<0.05), i. e. less myopic in exotropes and less hyperopic in esotropes. The prevalence of high anisometropia (difference in spherical equivalence between the two eyes of more than 2D) and astigmatism (astigmatism of more than 2D) increases as the eyes become more ametropic, especially prominent in exotropes. The presence of anisometropia and astigmatism may play a role in the pathogenesis of heterotropia.